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JCI—下丘脑内源性大麻素信号调节麻醉小鼠的觉醒
JCI—下丘脑内源性大麻素信号调节麻醉小鼠的觉醒
1.eCB modulation of arousal from anesthesia.
2.Electrophysiological and ultrastructural characteristics of DMH GABAergic and glutamatergic neurons.
3.Isoflurane-mediated increase of eCB signaling in DMH glutamatergic but not GABAergic synapses.
4.Blockade of CB1R at DMH glutamatergic synapses accelerates arousal through VLPO and Pef.
5.CB1R deletion from PFC glutamatergic neurons accelerates arousal.
6.PFC-DMH-Pef/VLPO inactivation counteracts AM281’s effects on arousal.
7.PFC-DMH-Pef/VLPO activation enhances arousal.
8.Proposed model for eCB modulation of brain circuitry participating in arousal from anesthesia.
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